Foods That Inflame Arthritis


ORLANDO, Fla. (Ivanhoe Newswire) — Nearly one in four people in the US are living with arthritis. It’s the leading cause of work disability, costing billions in lost earnings. Age, obesity, and genetics are risk factors that can lead to arthritis, but as Ivanhoe reports, what you eat can also cause your symptoms to worsen. Inflame arthritis

Achy, sore, and stiff joints are classic signs of arthritis and they don’t just affect the elderly.

Mathew Pombo, MD, Orthopedic Surgeon at Emory Orthopaedics & Spine Center says, “We’re seeing arthritis at an earlier age, not only in the knees, but shoulders, really everywhere.”

Arthritis can severely impact a person’s day to day.

Stephen Messier, PhD, Prof. of Health & Exercise Science at Wake Forest University says, “The main symptom is pain, decrease in quality of life, decrease in mobility.”

And often limit a person’s ability to work. According to the CDC, one in 10 adults have to limit their activities due to arthritis. But experts say you may be able to reduce the severity of your symptoms by avoiding certain arthritis-inflaming foods. Wheat products like pasta, bread, bagels and crackers contain gluten and it is a protein that can aggravate arthritic joints. In fact, studies have shown that people with celiac disease are at an increased risk of developing rheumatoid arthritis, or RA, but those who went gluten-free were able to alleviate their symptoms. Added sugars are another thing to avoid if you have arthritis. A study in New England found those who drank sugary beverages five times or more per week were three times more likely to have arthritis. Other foods that can increase your risk and worsen symptoms include red meats, processed foods, alcohol, and foods high in omega-six fats like vegetable oils. Helping you to live pain-free.

Your diet is not the only factor in arthritis pain. Experts say managing your smoking status, activity level, and body weight can also help ease arthritis symptoms.

Contributors to this news report include: Milvionne Chery, Producer; Bob Walko, Editor.



REPORT #3059

BACKGROUND: Arthritis is the swelling and tenderness of one or more joints. The main symptoms of arthritis are joint pain and stiffness, which typically worsen with age. The most common types of arthritis are osteoarthritis and rheumatoid arthritis. Osteoarthritis causes cartilage — the hard, slippery tissue that covers the ends of bones where they form a joint — to break down. Rheumatoid arthritis is a disease in which the immune system attacks the joints, beginning with the lining of joints. More than 22% of American adults (over 52.5 million people) have arthritis or another rheumatic condition diagnosed by a doctor. By 2030, the number of people with arthritis (adults 18 years and older) is expected to rise to 67 million.


DIAGNOSIS: The most common signs and symptoms of arthritis involve the joints. Depending on the type of arthritis, signs and symptoms may include: pain, stiffness, swelling, redness, and/or decreased range of motion. During the physical exam, doctors check your joints for swelling, redness and warmth. They’ll also want to see how well you can move your joints. The analysis of different types of body fluids can help pinpoint the type of arthritis you may have. Fluids commonly analyzed include blood, urine and joint fluid. To obtain a sample of joint fluid, doctors cleanse and numb the area before inserting a needle in the joint space to withdraw some fluid. These types of tests can detect problems within the joint that may be causing your symptoms. Examples include: X-rays, CT scan, MRI, and/or ultrasound.


NEW REGULATIONS: A research team, led by Professor Sung Ho Park in the Department of Biological Sciences at UNIST announced the results of a study on osteoblasts that damage joint bones in patients with rheumatoid arthritis. In this study, the research team studied the possibility of a treatment method targeting mechanisms related to the differentiation process of osteoblasts that melt bones through enzyme reactions. First, it was confirmed that a superinhancer is formed near the NFATC1 gene, which is known to be an important factor in the formation of osteoblasts, and it is formed only in osteoblasts. Through this study, it has been confirmed that NFATC1 super-in-hander RNA, which is formed during osteoblast differentiation, can be used as a treatment target. “[O]ur study is the first to identify SEs and SE-eRNAs in human osteoclasts and provides a better understanding of human osteoclast biology, thereby opening new therapeutic avenues for human pathological bone destruction,” noted the research team.


* For More Information, Contact:            Kimberly McGrath

Alysia Satchel

Free weekly e-mail on Medical Breakthroughs from Ivanhoe. To sign up: